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cadmium chloride (cdcl2)  (FUJIFILM)

 
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    FUJIFILM cadmium chloride (cdcl2)
    Cadmium Chloride (Cdcl2), supplied by FUJIFILM, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/cadmium+chloride+(cdcl2)/cdcl+2/10__1021_slash_acsapm__5c00381-46-28-34
    Average 90 stars, based on 1 article reviews
    cadmium chloride (cdcl2) - by Bioz Stars, 2026-10
    90/100 stars

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    Column Chromatography:

    Article Title: Location-selective immobilisation of single-atom catalysts on the surface or within the interior of ionic nanocrystals using coordination chemistry.
    Article Snippet: .. Cadmium chloride (CdCl2, 95%, FUJIFILM Wako), oleylamine (OAM, 70%, Sigma–Aldrich), n-octylamine (nOctNH2, 98.0%, FUJIFILM Wako), selenium (Se, 100 mesh, 99.99%, Sigma–Aldrich), tri-n-butylphosphine (TBP, 95.0%, TCI), ethanol (EtOH, 99.5%, Japan Alcohol Trading), n-hexane (95.0%, FUJIFILM Wako), toluene (99.0%, FUJIFILM Wako), methanol (MeOH, 99.5%, FUJIFILM Wako), chloroform (CHCl3, 99.0%, FUJIFILM Wako), activated basic alumina (Al2O3, for column chromatography, ca. .. 75 μm, pH 9.0–11.0 at 100 g/L, Wako), cis-dichlorobis(dimethyl sulfoxide)platinum(II) (cis-[PtCl2(dmso)2], 97%, Sigma–Aldrich), anhydrous acetonitrile (MeCN, 99.8%, super dehydrated, FUJIFILM Wako), cis-bis(benzonitrile)dichloroplatinum(II) (cis[PtCl2(NCPh)2], 98%, Sigma–Aldrich), cis-dichlorobis(triphenylphosphine)platinum(II) (cis-[PtCl2(PPh3)2], 8.6–9.3% Cl, Sigma–Aldrich), hexadecyltrimethylammonium chloride (CTAC, 98.0%, Sigma–Aldrich) potassium tetrachloroplatinate(II) (K2PtCl4, 98.0% Wako), dichloromethane (DCM, 99.0%, FUJIFILMWako), anhydrousmethanol (MeOH, 99.8%, super dehydrated, FUJIFILM Wako), acetic acid (AcOH, 99.7%, Wako), potassium hydroxide (KOH, 85.0%, FUJIFILM Wako), 11- mercaptoundecanoic acid (MUA, 95%, Sigma–Aldrich), cadmium bromide tetrahydrate (CdBr2·4H2O, 98%, FUJIFILM Wako), dimethyl sulfoxide (DMSO, 99.8%, Kishida), triethanolamine (TEOA, 99.0%, Sigma–Aldrich), hydrochloric acid (HCl aq, 35.0%–37.0%, for atomic absorption spectrometry, Wako), nitric acid (HNO3 aq, 60%–61%, for metal analysis, Wako), and boron nitride (spectral grade, Wako) were used as received without further purification.



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    (A) Eighteen Hu sheep were randomly divided into three groups: the control group (CON) receiving sterile saline, the cadmium-exposed group (Cd) administered 20 mg/kg CdCl₂ daily via oral gavage, and the therapeutic intervention group (CA) treated with combined 20 mg/kg CdCl₂ and 5 mg/kg sodium octanoate. All animals were euthanized for tissue collection on day 22 post-modeling ( n = 6). (B) Body weight change trends in Hu sheep of CON, Cd and CA groups. (C) The morphological characteristics of H&E staining of the ileum tissues in the three experimental groups, as well as the quantitative statistics of villus length. The inflammatory infiltration areas in the Cd group are indicated by red arrows. (D) The PAS staining of the ileum tissues of the three groups of Hu sheep and the quantitative statistics of the number of goblet cells in the crypts. (E) MUC2 immunohistochemical staining demonstrated goblet cell distribution and secretory status in ileal tissues across experimental groups. Quantitative statistics of the expression levels of the number of positive cells. (F) The cadmium-exposed group (Cd) exhibited marked elevation in IL-1 β , IL-6, and TNF- α levels, indicative of systemic inflammatory escalation. Sodium octanoate intervention (CA group) significantly attenuated these pro-inflammatory factors expression level (* p < 0.05, ** p < 0.01, *** p < 0.001).
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    (A) Eighteen Hu sheep were randomly divided into three groups: the control group (CON) receiving sterile saline, the cadmium-exposed group (Cd) administered 20 mg/kg CdCl₂ daily via oral gavage, and the therapeutic intervention group (CA) treated with combined 20 mg/kg CdCl₂ and 5 mg/kg sodium octanoate. All animals were euthanized for tissue collection on day 22 post-modeling ( n = 6). (B) Body weight change trends in Hu sheep of CON, Cd and CA groups. (C) The morphological characteristics of H&E staining of the ileum tissues in the three experimental groups, as well as the quantitative statistics of villus length. The inflammatory infiltration areas in the Cd group are indicated by red arrows. (D) The PAS staining of the ileum tissues of the three groups of Hu sheep and the quantitative statistics of the number of goblet cells in the crypts. (E) MUC2 immunohistochemical staining demonstrated goblet cell distribution and secretory status in ileal tissues across experimental groups. Quantitative statistics of the expression levels of the number of positive cells. (F) The cadmium-exposed group (Cd) exhibited marked elevation in IL-1 β , IL-6, and TNF- α levels, indicative of systemic inflammatory escalation. Sodium octanoate intervention (CA group) significantly attenuated these pro-inflammatory factors expression level (* p < 0.05, ** p < 0.01, *** p < 0.001).
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    cadmium chloride (cdcl2)  (FUJIFILM)
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    FUJIFILM cadmium chloride (cdcl2)
    (A) Eighteen Hu sheep were randomly divided into three groups: the control group (CON) receiving sterile saline, the cadmium-exposed group (Cd) administered 20 mg/kg CdCl₂ daily via oral gavage, and the therapeutic intervention group (CA) treated with combined 20 mg/kg CdCl₂ and 5 mg/kg sodium octanoate. All animals were euthanized for tissue collection on day 22 post-modeling ( n = 6). (B) Body weight change trends in Hu sheep of CON, Cd and CA groups. (C) The morphological characteristics of H&E staining of the ileum tissues in the three experimental groups, as well as the quantitative statistics of villus length. The inflammatory infiltration areas in the Cd group are indicated by red arrows. (D) The PAS staining of the ileum tissues of the three groups of Hu sheep and the quantitative statistics of the number of goblet cells in the crypts. (E) MUC2 immunohistochemical staining demonstrated goblet cell distribution and secretory status in ileal tissues across experimental groups. Quantitative statistics of the expression levels of the number of positive cells. (F) The cadmium-exposed group (Cd) exhibited marked elevation in IL-1 β , IL-6, and TNF- α levels, indicative of systemic inflammatory escalation. Sodium octanoate intervention (CA group) significantly attenuated these pro-inflammatory factors expression level (* p < 0.05, ** p < 0.01, *** p < 0.001).
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    (A) Eighteen Hu sheep were randomly divided into three groups: the control group (CON) receiving sterile saline, the cadmium-exposed group (Cd) administered 20 mg/kg CdCl₂ daily via oral gavage, and the therapeutic intervention group (CA) treated with combined 20 mg/kg CdCl₂ and 5 mg/kg sodium octanoate. All animals were euthanized for tissue collection on day 22 post-modeling ( n = 6). (B) Body weight change trends in Hu sheep of CON, Cd and CA groups. (C) The morphological characteristics of H&E staining of the ileum tissues in the three experimental groups, as well as the quantitative statistics of villus length. The inflammatory infiltration areas in the Cd group are indicated by red arrows. (D) The PAS staining of the ileum tissues of the three groups of Hu sheep and the quantitative statistics of the number of goblet cells in the crypts. (E) MUC2 immunohistochemical staining demonstrated goblet cell distribution and secretory status in ileal tissues across experimental groups. Quantitative statistics of the expression levels of the number of positive cells. (F) The cadmium-exposed group (Cd) exhibited marked elevation in IL-1 β , IL-6, and TNF- α levels, indicative of systemic inflammatory escalation. Sodium octanoate intervention (CA group) significantly attenuated these pro-inflammatory factors expression level (* p < 0.05, ** p < 0.01, *** p < 0.001).

    Journal: Frontiers in Veterinary Science

    Article Title: Cadmium induced ferroptosis and inflammation in sheep via targeting ACSL4/NF-κB axis

    doi: 10.3389/fvets.2025.1617190

    Figure Lengend Snippet: (A) Eighteen Hu sheep were randomly divided into three groups: the control group (CON) receiving sterile saline, the cadmium-exposed group (Cd) administered 20 mg/kg CdCl₂ daily via oral gavage, and the therapeutic intervention group (CA) treated with combined 20 mg/kg CdCl₂ and 5 mg/kg sodium octanoate. All animals were euthanized for tissue collection on day 22 post-modeling ( n = 6). (B) Body weight change trends in Hu sheep of CON, Cd and CA groups. (C) The morphological characteristics of H&E staining of the ileum tissues in the three experimental groups, as well as the quantitative statistics of villus length. The inflammatory infiltration areas in the Cd group are indicated by red arrows. (D) The PAS staining of the ileum tissues of the three groups of Hu sheep and the quantitative statistics of the number of goblet cells in the crypts. (E) MUC2 immunohistochemical staining demonstrated goblet cell distribution and secretory status in ileal tissues across experimental groups. Quantitative statistics of the expression levels of the number of positive cells. (F) The cadmium-exposed group (Cd) exhibited marked elevation in IL-1 β , IL-6, and TNF- α levels, indicative of systemic inflammatory escalation. Sodium octanoate intervention (CA group) significantly attenuated these pro-inflammatory factors expression level (* p < 0.05, ** p < 0.01, *** p < 0.001).

    Article Snippet: Cadmium chloride (CdCl2) and sodium octanoate were sourced from Macklin Biochemical (Shanghai, China).

    Techniques: Control, Sterility, Saline, Staining, Immunohistochemical staining, Expressing